Fistulas between the arteries and the gastrointestinal tract are rare but can be fatal. We present a case of an ilioenteric fistula between the left external iliac artery and sigmoid colon caused by radiotherapy for cervical cancer, which was treated with endovascular management using a stent graft. A 38-year-old woman underwent concurrent chemoradiotherapy for cervical cancer recurrence. Approximately 9 months later, the patient suddenly developed hematochezia. On her first visit to the emergency room of our hospital, computed tomography (CT) images did not reveal extravasation of contrast media. However, 8 hours later, she revisited the emergency room because of massive hematochezia with a blood pressure of 40/20 mmHg and a heart rate of 150 beats per minute. At that time, CT images showed the presence of contrast media in almost the entire colon. The patient was referred to the angiography room at our hospital for emergency angiography. Inferior mesenteric arteriography did not reveal any source of bleeding. Pelvic arteriography showed contrast media extravasation from the left external iliac artery to the sigmoid colon; this was diagnosed as an ilioenteric fistula and treated with a stent graft. When the bleeding focus is not detected on visceral angiography despite massive arterial bleeding, pelvic arteriography is recommended, especially in patients with a history of pelvic surgery or radiotherapy.
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Resection of sigmoid cancer with bladder invasion using laparoscopic combined with a cystoscopic holmium laser: an innovative surgical procedure Ronghua Wu, Cong Xu, Xing Liu, Weihua Fu, Yujia Chen, Jingzhen Zhu, Guangsheng Du Lasers in Medical Science.2023;[Epub] CrossRef
Lymphorrhea is a rare but potentially severe complication that occurs after various surgical procedures. Untreated lymphorrhea may lead to wound dehiscence, infection, and prolonged hospital stay. Currently, there is no standard effective treatment. Early management usually includes leg elevation, drainage, and pressure dressing. However, these methods are associated with prolonged recovery and high recurrence rates. We report a case of lymphorrhea from a calf wound after endoscopic great saphenous vein (GSV) harvesting for coronary artery bypass grafting (CABG). The patient presented with intractable oozing from the postoperative wound on the right calf. Lymphorrhea perGsisted for 6 weeks despite negative-pressure wound therapy with a long-acting somatostatin. We performed unilateral pedal lymphangiography that confirmed wound lymphorrhea, followed by glue embolization. No recurrence was observed after 8 months of follow-up. This case report demonstrates the successful use of lymphangiography with glue embolization in the control of lymphorrhea after GSV harvesting for CABG.
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Free Chimeric Anterolateral Thigh Flap with Vastus Lateralis Muscle Transfer for the Treatment of Intractable Upper Arm Lymphorrhea due to Large Upper Body Lymphangioma Sei Yoshida, Hideki Kadota, Kentaro Anan, Nobuaki Hatakeyama International Journal of Surgical Wound Care.2024; 5(2): 46. CrossRef
Supermicrosurgical lymphatic venous anastomosis for intractable lymphocele after great saphenous vein harvesting graft Hirofumi Imai, Shuhei Yoshida, Toshiro Mese, Solji Roh, Isao Koshima Journal of Vascular Surgery Cases, Innovations and Techniques.2022; 8(1): 45. CrossRef
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Lymphatic complications after harvesting venous conduits in coronary artery bypass grafting surgery D. V. Manvelyan, Yu. Y. Vechersky, V. V. Zatolokin, M. S. Kuznetsov, B. N. Kozlov The Siberian Journal of Clinical and Experimental Medicine.2021; 36(3): 27. CrossRef
Congenital abnormalities of the coronary arteries are found in 0.6% to 1.3% of patients in coronary angiography. Dual left anterior descending coronary artery (LAD) is a rare coronary anomaly and is incidentally detected during coronary angiography. We report a case of a 65-year-old female with a rare coronary anomaly who was diagnosed with dual LAD via coronary computed tomography and coronary angiography. The imaging studies revealed dual LAD originating from the left main stem and right coronary sinus. These angiographic findings were considered to be consistent with the type IV variety of dual LAD by Spindola-Franco classification. Recognition of dual LAD is important to prevent errors of interpretation of the coronary angiogram and for optimal surgery.
The left aortic arch with an aberrant right subclavian artery, or arteria lusoria, is the most common aortic arch anomaly, occurring in 0.5-2.5% of individuals. In such cases, the angular course of the arteria lusoria to the ascending aorta imposes difficulty in passing a guide wire to the ascending aorta during right transradial catheterization. Here, the case of a 53-year-old woman with intermittent chest tightness and coughing is reported. Aberrant right subclavian artery (arteria lusoria) was diagnosed via aortogram during right transradial coronary angiography. Compression of the esophagus and trachea by the aberrant right subclavian artery was demonstrated by chest computed tomography (CT).
Purpose:This study was done to arrange the cases showing temporally hyperperfusion in the periphery of the liver, to check the etiology and mechanism, and to find out the new radiologic role on diffuse liver disease.
Materials and Methods:We reviewed 12 cases of showing transient arterial hyperperfusion in the just peripheral portion of the liver on the arterial dominant phase on dynamic CT and the absence of abnormal perfusion between central and peripheral portion on tissue equilibrium phase. We retrospectively analyzed final diagnosis and the presence of main portal vein thrombi, and cavernous transformation.
Results :Final diagnosis in 12 cases was as follows: diffuse liver disease was in seven cases, pancreatitis in three and pyogenic portal thrombosis in two. Main portal vein thrombosis were detected all cases of pancreatitis and pyogenic portal thrombosis. In seven diffuse liver disease, two cases shown thrombosed, two cases shown normal and the other cases are collapsed. Cavernous transformation was in three cases of pancreatitis and two of diffuse liver disease.
Conclusion :We think that the causes of this phenomenon maybe as follows: the difference of the hemodynamic compensation mechanism between central and peripheral zone of the liver, presence of microscopic thrombi in peripheral portal branch which cannot be detected by imaging technique, hypercoagulability in portal area, the systematic destruction of terminal portal branch and the development of ectopic portal pathway.